Risk factors for acute bilirubin encephalopathy on admission to two Myanmar national paediatric hospitals

Table 5 Selected characteristics and outcomes of neonates with and without ABE at admission

		Hospital A					Hospital B
		ABE at admission (n = 32)		No ABE at admission (n = 219)		p-value	ABE at admission (n = 72)		No ABE at admission (n = 267)		P-value
		n	%	n	%	p-value	n	%	n	%	P-value
Demographic	Male^a	17	53.1	125	57.1	0.71	45	62.5	150	57.9	0.50
	Home birth	22	68.8	56	25.6	<0.0001	38	52.8	50	18.7	<0.0001
	Referred from home	16	50.0	38	17.4	0.0001	40	55.6	50	18.7	<0.0001
Selected risk factors for hyperbilirubinaemia	Preterm (<37 weeks’)^b	3	9.4	35	16.1	0.43	6	8.3	79	30.3	<0.0001
	Previous sibling	0	0.0	1	0.5	1.0	15	20.8	51	19.1	0.74
	Significant bruising	1	3.1	1	0.5	0.24	3	4.2	6	2.3	0.41
Previous phototherapy		2	6.3	26	11.9	0.55	9	12.5	25	10.6	0.67
Suspected cause (multiple selections permitted)	Suspected infection	23	71.9	135	61.6	0.32	34	47.2	149	55.8	0.23
	G6PD deficiency^c	14	43.8	77	35.2	0.43	29	40.3	29	10.9	<0.0001
	ABO incompatibility	3	9.4	40	18.3	0.31	8	11.1	44	16.5	0.36
	Rhesus incompatibility	0	0.0	4	1.8	1.0	0	0.0	2	0.8	1.0
	Other/Not specified	3	9.4	11	5.0	0.40	4	5.6	28	10.5	0.26
Discharge Status	Died	15	46.9	3	1.4	<0.0001	18	25.0	20	7.5	<0.0001
	Lived, with ABE	5	15.6	0	0.0		5	6.9	0	0.0
	All other discharge	12	37.5	216	98.6		49	68.1	247	92.5

n number, IQR Interquartile range (25^th percentile – 75^th percentile)
^aGender missing for 8 records at Hospital B (none of whom had ABE at presentation)
^bGestation missing for two records at Hospital A, and six records at Hospital B (none of the eight had ABE at presentation)
^cScreening test performed, and assessed as screen positive

ISSN: 2054-958X